雜志介紹

Circulation-heart Failure雜志介紹

《Circulation-heart Failure》是一本以English為主的未開放獲取國際優(yōu)秀期刊，中文名稱循環(huán)心力衰竭，本刊主要出版、報道醫(yī)學(xué)-CARDIAC & CARDIOVASCULAR SYSTEMS領(lǐng)域的研究動態(tài)以及在該領(lǐng)域取得的各方面的經(jīng)驗和科研成果，介紹該領(lǐng)域有關(guān)本專業(yè)的最新進展，探討行業(yè)發(fā)展的思路和方法，以促進學(xué)術(shù)信息交流，提高行業(yè)發(fā)展。該刊已被國際權(quán)威數(shù)據(jù)庫SCIE收錄，為該領(lǐng)域相關(guān)學(xué)科的發(fā)展起到了良好的推動作用，也得到了本專業(yè)人員的廣泛認可。該刊最新影響因子為7.8，最新CiteScore 指數(shù)為12.9。

本刊近期中國學(xué)者發(fā)表的論文主要有：

• Characteristics and Outcome of Patients With a History of Cancer Undergoing Durable Left Ventricular Assist Device Implantation

  Author: Tie, Hongtao; Zhu, Jing; Akin, Sakir; Allen, Larry A.; Huang, Bi; Martens, Sabrina; Welp, Henryk; Simpkin, Andrew; Shi, Rui; Wu, Qingchen; Soliman, Osama; Caliskan, Kadir; Chen, Dan

• Baseline Characteristics of Pediatric Patients With Heart Failure Due to Systemic Left Ventricular Systolic Dysfunction in the PANORAMA-HF Trial

  Author: Shaddy, Robert; Burch, Michael; Kantor, Paul F.; Solar-Yohay, Susan; Garito, Tania; Zhang, Sijia; Kocun, Michele; Bonnet, Damien

• Sympathoexcitation in Rats With Chronic Heart Failure Depends on Homeobox D10 and MicroRNA-7b Inhibiting GABBR1 Translation in Paraventricular Nucleus.

  Author: Wang R, Huang Q, Zhou R, Dong Z, Qi Y, Li H, Wei X, Wu H, Wang H, Wilcox CS, Hultstr?m M, Zhou X, Lai EY.

• Transcription Factor 7-like 2 Mediates Canonical Wnt/β-Catenin Signaling and c-Myc Upregulation in Heart Failure.

  Author: Hou N, Ye B, Li X, Margulies KB, Xu H, Wang X, Li F.

英文介紹

Circulation-heart Failure雜志英文介紹

Circulation: Heart Failure considers for publication content related to heart failure, mechanical circulatory support and heart transplant science and medicine, including studies conducted in humans or analyses of human data, as well as preclinical studies that have direct clinical correlation or relevance. As a primarily clinical journal, few basic and preclinical studies are ultimately published. Those that will be considered are novel, and significantly and directly advance the field of heart failure. The editors are especially interested in basic science studies directly related to heart failure that are highly informative and translatable to human disease and are conducted in vertebrate models. Less than 15% of submitted articles are ultimately published. Work not directly involving heart failure or heart failure therapies will not be considered, including work on mechanisms of myocardial infarction, reperfusion injury, or arrhythmia in the absence of heart failure substrate.